Although additional investigation will be had a need to elucidate mechanistic details, our data as well as various other studies indeed support the probability of post-transcriptional or post-translational mechanisms mediated with the sialyltransferase ST6GAL1 in controlling the protein level and function of POU5F1 in hPSCs. ST6GAL1 knockdown affected mobile reprogramming, reducing the real amount of … Continue reading Although additional investigation will be had a need to elucidate mechanistic details, our data as well as various other studies indeed support the probability of post-transcriptional or post-translational mechanisms mediated with the sialyltransferase ST6GAL1 in controlling the protein level and function of POU5F1 in hPSCs